Management of children with Guillain-Barre syndrome. Jones HR Jr. Guillain-Barre syndrome: perspectives with infants and children. Semin Pediatr Neurol ; 7 2 : Beghi E et al. Guillain-Barre syndrome.

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Management of children with Guillain-Barre syndrome. Jones HR Jr. Guillain-Barre syndrome: perspectives with infants and children. Semin Pediatr Neurol ; 7 2 : Beghi E et al. Guillain-Barre syndrome. Clinicoepidemiologic features and effect of infl uenza vaccine. Arch Neurol ; 42 11 : Occurrence, clinical manifestations, and prognosis of Guillain-Barre syndrome. Arch Dis Child ; 66 6 : ; discussion Olive JM et al. Epidemiologic study of Guillain-Barre syndrome in children Yuki N et al. Carbohydrate mimicry between human ganglioside GM1 and Campylobacter jejuni lipooligosaccharide causes Guillain-Barre syndrome.

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Serum anti-GQ1b IgG antibody is associated with ophthalmoplegia in Miller Fisher syndrome and Guillain- Barre syndrome: clinical and immunohistochemical studies. Neurology ; 43 10 : Seroreactivity to Campylobacter jejuni and gangliosides in patients with Guillain- Barre syndrome. J Infect Dis ; 4 : Hao Q et al. Anti-GalNAc-GD1a antibody-associated Guillain- Barre syndrome with a predominantly distal weakness without cranial nerve impairment and sensory disturbance.

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Síndrome de Guillain-Barré

The demyelinating form of Guillain-Barre syndrome destroys the protective covering of the peripheral nerves myelin sheath , preventing the nerves from transmitting signals to the brain. Weakness and tingling in your extremities are usually the first symptoms. These sensations can quickly spread, eventually paralyzing your whole body. In its most severe form Guillain-Barre syndrome is a medical emergency. Most people with the condition must be hospitalized to receive treatment. The exact cause of Guillain-Barre syndrome is unknown. But two-thirds of patients report symptoms of an infection in the six weeks preceding.

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Guillain–Barré syndrome

Although the cause is unknown, the underlying mechanism involves an autoimmune disorder in which the body's immune system mistakenly attacks the peripheral nerves and damages their myelin insulation. In those with severe weakness, prompt treatment with intravenous immunoglobulins or plasmapheresis , together with supportive care, will lead to good recovery in the majority of people. This is followed by weakness of the legs and arms that affects both sides equally and worsens over time. The plateau phase can take between two days and six months, but the most common duration is a week. This may consist of upper respiratory tract infection rhinitis, sore throat or diarrhea.

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